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ePub Plant mitochondria: With emphasis on RNA editing and cytoplasmic male sterility download

ePub Plant mitochondria: With emphasis on RNA editing and cytoplasmic male sterility download
ISBN13:
978-1560817673
ISBN:
1560817674
Publisher:
VCH (1993)
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ePub file:
1312 kb
Fb2 file:
1715 kb
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4.7
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Keywords: Mitochondria. Cytoplasmic male sterility (CMS). Restoration of fertility (Rf). Cytoplasmic male sterility of rice with boro II cytoplasm is. caused by a cytotoxic peptide and is restored by two related PPR motif genes via dis-. tinct modes of mRNA silencing. Plant Cell 18, 676–687. Pentatricopeptide repeat (PPR). Restoration of fertility complex (RFC). Mitochondria are essential organelles in cells not only because they supply over 90% of the cell's energy but also. because their dysfunction is associated with disease. Owing to the importance of mitochondria, there are many. Wang, . Jiang, . Li, . Zhang, . Guan, . 2012. Complete sequence of.

Cytoplasmic male sterility is total or partial male sterility in plants as the result of specific nuclear and mitochondrial interactions. Male sterility is the failure of plants to produce functional anthers, pollen, or male gametes. Joseph Gottlieb Kölreuter was the first to document male sterility in plants. He reported anther abortion within species and specific hybrids.

Nuclear restoration of cytoplasmic male sterility in sunflower is associated with the tissue-specific regulation of a novel mitochondrial gene.

Nuclear restoration of cytoplasmic male sterility in sunflower is associated with the tissue-specific regulation of a novel mitochondrial gene. PubMedGoogle Scholar.

Cytoplasmic male sterility (CMS) is a mysterious natural phenomenon, and the mechanism of the origin of CMS is unknown. Despite successful utilization of CMS and restoration of fertility (Rf) in practice, the underlying mechanisms of these processes remain elusive

Cytoplasmic male sterility (CMS) is a mysterious natural phenomenon, and the mechanism of the origin of CMS is unknown. Despite successful utilization of CMS and restoration of fertility (Rf) in practice, the underlying mechanisms of these processes remain elusive. Download full-text PDF. Source.

Here, we show that in mitochondria from the sterile line, this novel ORF (ORF522) is cotranscribed with atpA. Northern blot analysis using RNA from both seedlings and male florets shows a flower-specific reduction in the level of the ORF522 transcript in the restored hybrid line. We have identified the product of the ORF522 as being a 15 kDa protein previously observed in sterile plant mitochondria by in organello translation. The reduction is not due to a reduced transcription rate as demonstrated by 'run-on' experiments using mitochondria isolated from male florets.

Cytoplasmic male sterility (CMS) is the maternally inherited inability of a plant to produce or shed viable pollen. CMS is conditioned by gain-of-function mitochondrial genome mutations, giving rise to novel mitochondrial proteins and facilitating the use of CMS in hybrid crop seed production. A mitochondrial mutation is transmitted to all progeny of a cross, which allows the breeder to maintain the male sterility trait without the inconvenience of gene segregation each generation.

Cytoplasmic Genetic male sterility This type of male sterility systems is common in many plant species across plant kingdom. The sterility is manifested by the influence of both nuclear and cytoplasmic genes. There are commonly two types of cytoplasms, N (normal) and S (sterile).

Male sterile plants were also obtained by expressing specifically a bacterial ribonuclease in the anthers. The economical benefits of artificially engineered male-sterile plants or carrying the (native) spontaneous CMS phenotype, implies the restoration to obtain fertile hybrids that will be used in agriculture.